Eva Poveda

Dr. Eva Poveda is the Research Lead of the Group of Virology and Pathogenesis at the Galicia Sur Health Research Institute (IISGS) (http://www.iisgaliciasur.es/research-areas/inflammatory-and-infectious-diseases-immune-disorders/virology-pathogenesis/?lang=en). Her research career is focused on the study of HIV/AIDS and viral hepatitis since 2001. The results of her work have mainly focused on the molecular epidemiology, drug resistance, and pathogenesis of these infections, and have had direct applicability on the clinical management of patients. She has published 140 articles (h-index: 32) in major international scientific journals (https://www.ncbi.nlm.nih.gov/pubmed/?term=poveda+e). Dr. Poveda also has extensive experience in leading national and international projects in HIV research in HV, mainly in Europe and in the USA at Case Western Reserve University School of Medicine (CWRU) in Cleveland, Ohio. She has co-authored several guidelines on the treatment of HIV and HCV infections, and the clinical management of HIV Drug Resistance (i.e. GESIDA, GEHEP, ESAR, HIV/AIDS Network of Spain). She is currently the co-leader of the Working Group on HIV Drug Resistance in the Spanish HIV/AIDS Network (GESIDA), and lead author of the current 2018 Spanish guidelines on HIV drug resistance (GeSIDA). She is also actively engaged in scientific teaching and has directed 6 doctoral theses: 3 at the Universidad Complutense de Madrid and 3 at Universidade de A Coruña (summa cum laude). Dr. Poveda also actively participates in projects and initiatives for Science Communication to bring scientific knowledge to the general public, as well as promoting women in science. Her scientific contributions have been recognized with several awards, including the 2012 LOREAL-UNESCO Award for Women in Science Program in Spain. Dr. Poveda has established a solid collaboration with the Laboratory of Interesting Immunology led by Dr. Michael Lederman at Case Western Reserve University (Cleveland, Ohio), in order to study the significance of extracellular vesicles (EVs) and mitocondria in the pathogenesis of HIV disease.